2016 VAM: 1206 - Inflammatory Origins of CRS: Uncovering Opportunities for Disease Prevention and Modification
The goal of this session is to explore the role of Inflammation in Chronic Rhinosinusitis. As we move away from an infectious etiology as the primary trigger for sinus disease and more toward a complex model if multiple inflammatory pathways, we hope to move this topic toward the practicing clinician.
The mucosal/bacterial interface, whether in a biofilm or as a result of the microbiome is thought to drive much of this inflammation. Additionally, as the role of non-antimicrobial therapy increases for CRS, we will explore some of the new inflammatory pathways and look at the emerging therapies for CRS.
AMA PRA Category 1 Credits™: 2.50
Credit must be claimed by May 31, 2018. Any credit request on or after June 1, 2018 will be subject to an administrative fee.
Allied Health Professionals
Upon completion of this session, participants should be able to:
1. Describe the role of the microbiome and biofilms in the development of CRS
2. Discuss the implications of antibiotic use and overuse in CRS
3. Identify inflammatory pathways in CRS and opportunities for disease modification
Claus Bachert, MD PhD, Universitair Ziekenhuis Gent, Ghent, Belgium
Robert C. Kern, MD, Northwestern University Feinberg School of Medicine, Chicago, IL
Anju T. Peters, MD, Northwestern University Feinberg School of Medicine, Chicago, IL
Martin Wagenmann, MD FAAAAI, Henrich Heine University, Duesseldorf, Germany
- 2.50 AttendanceAttendance credit.
- 2.50 CECE credit.
- 2.50 CMECME credit.