This symposium will highlight multiple pathways by which LTC4, LTD4 and LTE4 promote type 2 immunopathology.
LEARNING OBJECTIVES:
Upon completion of this activity, participants should be able to:
- Recognize that each cysteinyl leukotriene has a distinct receptor preference and cell target
- Recognize the role of cysLTs as polyclonal activators of T cells and innate lymphoid cells Identify the epithelial targets of the cysLTs and their role in type 2 immunity
Moderator Introduction
David H. Broide, MB ChB FAAAAI
University of CA - San Diego
GPR99 Dependent Epithelial Homeostasis
Lora G. Bankova, MD
Brigham and Women
Leukotriene-Driven Platelet Activation Promotes IL-33-Dependent Immunopathology
Joshua A. Boyce, MD FAAAAI
Brigham and Women's Hospital
Activation of ILC2 Cells by Cysteinyl Leukotrienes
Taylor A. Doherty, MD FAAAAI
UC San Diego
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